JC Jennette, H Xiao, R Falk… - Contrib Nephrol, 2011 - content.karger.com Abstract Antineutrophil cytoplasmic autoantibodies (ANCA) are closely associated with systemic small vessel vasculitis characterized by segmental vessel wall necrotizing inflam- mation and a paucity of immunoglobulin deposition. Clinically, in vitro and experimental animal ... Cited by 1 - Related articles - All 3 versions
J Bontscho, A Schreiber, RA Manz… - Journal of the …, 2011 - Am Soc Nephrol Anti-neutrophil cytoplasmic autoantibodies (ANCA) cause vasculitis and necrotizing crescentic glomerulonephritis (NCGN). Steroids and cytotoxic drugs reduce mortality but can cause significant adverse events. The proteasome inhibitor bortezomib (BTZ) prevents glomerulonephritis ... All 5 versions
G Moroni, C Casati, S Quaglini, B Gallelli… - …, 2011 - journals.lww.com Wolters Kluwer Health may email you for journal alerts and information, but is committed to maintaining your privacy and will not share your personal information without your express consent. For more information, please refer to our Privacy Policy. ... Skip Navigation Links Home > Current ... Related articles - All 4 versions
D Odobasic, PY Gan, SA Summers… - The American Journal of …, 2011 - Elsevier T helper (Th)17 cells might contribute to immune-mediated renal injury. Thus, we sought to define the time course of IL-17A-induced kidney damage and examined the relation between Th17 and Th1 cells in a model of crescentic anti-glomerular basement membrane ... All 2 versions
S Guo, TA Wietecha, KL Hudkins, Y Kida… - Kidney …, 2011 - nature.com Mice transgenic for thymic stromal lymphopoietin (TSLP), under regulation of the lymphocyte-specific promoter Lck, develop cryoglobulinemia and membranoproliferative glomerulonephritis (MPGN) similar to the disease in patients. To determine whether infiltrating macrophages, ...
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