[PDF] from hindawi.comA Drusco, Y Pekarsky, S Costinean… - Lung, 2011 - downloads.hindawi.com 1 Department of Molecular Virology, Immunology, and Medical Genetics, The Ohio State University, Columbus, OH 43210, USA 2 Clinical Pathology, Regina Elena Institute, IFO, 00144 Rome, Italy 3 Department of Immunology and Cancer Research-IMRIC, The Lautenberg ... Related articles - View as HTML - All 4 versions
[HTML] from nih.govLV O'Keefe, A Colella, S Dayan… - Human molecular …, 2011 - Oxford Univ Press Common chromosomal fragile sites FRA3B and FRA16D are frequent sites of DNA instability in cancer, but their contribution to cancer cell biology is not yet understood. Genes that span these sites (FHIT and WWOX, respectively) are often perturbed (either increased or ... Cited by 2 - Related articles - All 7 versions
A Blumrich, M Zapatka, LM Brueckner… - Human molecular …, 2011 - Oxford Univ Press Common fragile sites (cFS) represent chromosomal regions that are prone to breakage after partial inhibition of DNA synthesis. Activation of cFS is associated with various forms of DNA instability in cancer cells, and is thought to be an initiating event in the generation of DNA ... Related articles - All 5 versions
A Letessier, GA Millot, S Koundrioukoff… - Nature, 2011 - nature.com Common fragile sites have long been identified by cytogeneticists as chromosomal regions prone to breakage upon replication stress 1 . They are increasingly recognized to be preferential targets for oncogene-induced DNA damage in pre-neoplastic lesions 2 and hotspots for ... Cited by 5 - Related articles - All 5 versions
E Ozeri-Galai, R Lebofsky, A Rahat, AC Bester… - Molecular Cell, 2011 - Elsevier ... Here, we show that even under normal growth conditions, replication fork progression along the fragilesite, FRA16C, is slow and forks frequently stall at AT-rich sequences, leading to activation of additional origins to enable replication completion. ... All 2 versions
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