R Müller… - Clinical Trials in Rheumatology, 2011 - Springer Trial Daily and alternate-day corticosteroid regimens in treatment of giant cell arteritis: comparison in a prospective study Substance All groups: 20 mg prednisone every 8 h for 5 days, then group A: 15 mg of prednisone every 8 h (n = 20) group B: 45 mg of prednisone every morning (n ...
N Hassan, B Dasgupta… - BMJ, 2011 - bmj.com A systematic review analysed the presenting clinical features in a mixture of studies with a total of 1435 cases of giant cell arteritis.3 The sensitivity of individual clinical features was relatively low (table⇓), reflecting the diverse presentation of this condition: 24% of cases had no ... Cited by 2 - Related articles - All 3 versions
KSM van der Geest, WH Abdulahad… - Annals of the …, 2011 - ard.bmj.com Background and objectives T cells are considered major players in the development of giant cell arteritis (GCA). To date most attention has been focused on Th1 and Th17 cells. However, the contribution of senescent T cells to this strictly ageing-associated vasculitis ...
SS Hayreh - Ischemic Optic Neuropathies, 2011 - Springer PMR was first described as “senile rheumatic gout” and named PMR in 1957 [1]. Compared with GCA, PMR is much more common [2, 3]. PMR is characterized by the presence of pain and stiffness in the neck, shoulders and hips, weight loss, low-grade fever, fatigue and ele- vated ...
SS Hayreh - Ischemic Optic Neuropathies, 2011 - Springer This has been a controversial topic for a long time and requires a detailed historical discussion because it has important implications. Originally visual loss due to GCA was thought to be due to central retinal artery occlusion (CRAO). Meadows in 1954 [1], however, found that in ...
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