MAW Rowh, A DeMicco, JE Horowitz, B Yin… - Oncogene, 2011 - nature.com Inactivating Tp53 mutations are frequent genetic lesions in human tumors that harbor genomic instability, including B lineage lymphomas with IG translocations. Antigen receptor genes are assembled and modified in developing lymphocytes by RAG/AID-initiated genomic ... All 3 versions
D Biswas, TA Milne, V Basrur… - Proceedings of the …, 2011 - National Acad Sciences A number of acute leukemias arise from fusion of the mixed lineage leukemia 1 protein (MLL) N terminus to a variety of fusion partners that have been reported to reside in one or more poorly defined complexes linked to transcription elongation through in- teractions with the ...
MT Palmer, YK Lee, CL Maynard, JR Oliver… - Journal of Biological …, 2011 - ASBMB Vitamin D deficiency is implicated in autoimmune disease. We therefore evaluated the effects of 1α,25-dihydroxyvitamin D 3 (1,25-D 3 ), the active form of vitamin D, on the development of T helper 1 (Th1), Th17, and Th9 cells, which are implicated in the pathogenesis of ... Cited by 3 - Related articles - All 4 versions
L Panman, E Andersson, Z Alekseenko… - Cell stem …, 2011 - ncbi.nlm.nih.gov The generation of specific types of neurons from stem cells offers important opportunities in regenerative medicine. However, future applications and proper verification of cell identities will require stringent ways to generate homogeneous neuronal cultures. Here we show that transcription ... Cited by 1
R Monteiro, C Pouget… - The EMBO Journal, 2011 - nature.com Lineage fate decisions underpin much of development as well as tissue homeostasis in the adult. A mechanistic paradigm for such decisions is the erythroid versus myeloid fate decision controlled by cross-antagonism between gata1 and pu.1 transcription factors. In this ... Cited by 1 - Related articles - All 3 versions
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