KVR Rao, PVB Reddy, KM Curtis… - Journal of …, 2011 - liebertonline.com To cite this article: Kakulavarapu V. Rama Rao, Pichili VB Reddy, Kevin M. Curtis and Michael D. Norenberg. Journal of Neurotrauma. March 2011, 28(3): 371-381. doi:10.1089/neu.2010.1705 . ... 1 Department of Pathology, University of Miami Miller School of Medicine, Miami, ... Related articles
AR Jayakumar, KS Panickar… - Journal of …, 2011 - Wiley Online Library This is an Accepted Article that has been peer-reviewed and approved for publication in the Journal of Neurochemistry, but has yet to undergo copy-editing and proof correction. Please cite this article as an “Accepted Article”; doi: 10.1111/j.1471-4159.2011.07211.x Related articles - All 4 versions
Y Bao, HM Bramlett, CM Atkins… - Journal of …, 2011 - liebertonline.com To cite this article: Ying-hui Bao, Helen M. Bramlett, Coleen M. Atkins, Jessie S. Truettner, George Lotocki, Ofelia F. Alonso, W. Dalton Dietrich. Journal of Neurotrauma. January 2011, 28(1): 35-42. doi:10.1089/neu.2010.1383. ... 1 Department of Neurological Surgery, ... Cited by 1 - Related articles - All 2 versions
AA Oliva Jr, Y Kang, JS Truettner… - Neuroscience, 2011 - Elsevier Edema, the accumulation of excess fluid, is a major pathological change in the brain that contributes significantly to pathology and mortality after moderate to severe brain injury. Edema is regulated by aquaporin (AQP) channels which transport water across cellular membranes. Six ... Cited by 1 - All 2 versions
RKV Rama, PV Reddy, KM Curtis… - Journal of …, 2011 - ncbi.nlm.nih.gov The development of cytotoxic brain edema resulting in increased intracranial pressure is a major cause of death occurring in the early phase of traumatic brain injury (TBI). Such edema predominantly develops as a consequence of astrocyte swelling. We recently documented that fluid ...
KVR Rao, PVB Reddy, KM Curtis… - Journal of …, 2011 - liebertonline.com To cite this article: Kakulavarapu V. Rama Rao, Pichili VB Reddy, Kevin M. Curtis and Michael D. Norenberg. Journal of Neurotrauma. March 2011, 28(3): 371-381. doi:10.1089/neu.2010.1705 . ... 1 Department of Pathology, University of Miami Miller School of Medicine, Miami, ... Related articles
AR Jayakumar, KS Panickar… - Journal of …, 2011 - Wiley Online Library This is an Accepted Article that has been peer-reviewed and approved for publication in the Journal of Neurochemistry, but has yet to undergo copy-editing and proof correction. Please cite this article as an “Accepted Article”; doi: 10.1111/j.1471-4159.2011.07211.x Related articles - All 4 versions
Y Bao, HM Bramlett, CM Atkins… - Journal of …, 2011 - liebertonline.com To cite this article: Ying-hui Bao, Helen M. Bramlett, Coleen M. Atkins, Jessie S. Truettner, George Lotocki, Ofelia F. Alonso, W. Dalton Dietrich. Journal of Neurotrauma. January 2011, 28(1): 35-42. doi:10.1089/neu.2010.1383. ... 1 Department of Neurological Surgery, ... Cited by 1 - Related articles - All 2 versions
AA Oliva Jr, Y Kang, JS Truettner… - Neuroscience, 2011 - Elsevier Edema, the accumulation of excess fluid, is a major pathological change in the brain that contributes significantly to pathology and mortality after moderate to severe brain injury. Edema is regulated by aquaporin (AQP) channels which transport water across cellular membranes. Six ... Cited by 1 - All 2 versions
RKV Rama, PV Reddy, KM Curtis… - Journal of …, 2011 - ncbi.nlm.nih.gov The development of cytotoxic brain edema resulting in increased intracranial pressure is a major cause of death occurring in the early phase of traumatic brain injury (TBI). Such edema predominantly develops as a consequence of astrocyte swelling. We recently documented that fluid ...
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