[HTML] from nih.govWL Holland, BT Bikman, LP Wang… - The Journal of clinical …, 2011 - ncbi.nlm.nih.gov 1 Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas, USA. 2 Program in Cardiovascular and Metabolic Diseases, Duke-National University of Singapore Graduate Medical School, Singapore. 3 Sarah W. Stedman Nutrition and ... Cited by 3 - Related articles - All 5 versions
P Perez-Martinez, J Delgado-Lista… - … American Journal of …, 2011 - Am Soc Nutrition 1 From the Lipid and Atherosclerosis Unit, IMIBIC/Reina Sofia University Hospital/University of Cordoba, and CIBER Fisiopatologia Obesidad y Nutricion, Instituto de Salud Carlos III, Córdoba, Spain (PP-M, JD-L, AG-R, and JL-M); the Nutrigenomics Research Group, ... Related articles - All 3 versions
I Dahlman - The American journal of clinical nutrition, 2011 - Am Soc Nutrition The effect of nutrient composition on human adipose gene expression, independently of body mass index, is largely unknown. It was therefore with great interest that I read the article by van Dijk et al (1) in a recent issue of the Journal. In their article, van Dijk et al report that ... Related articles - All 4 versions
MD Mantzaris, EV Tsianos… - The FEBS journal, 2011 - ncbi.nlm.nih.gov The aim of the present study was to investigate in detail the molecular mechanisms by which free fatty acids induce liver toxicity in liver cells. HepG2 and Huh7 human liver cell lines were exposed to varying concentrations of stearate (18:0), oleate (18:1), or mixtures of the two ... Cited by 1 - Related articles
M Sharma, F Urano… - Journal of Hepatology, 2011 - Elsevier The current model for JNK activation by SFA involves endoplasmic reticulum (ER) stress, which induces JNK activation through an inositol requiring enzyme 1 (IRE1αApoptosis Regulating Kinase 1 (ASK1) dependent mechanism. Here, we find that SFA-induced JNK activation is not ... All 3 versions
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